Monday, May 14, 2018

What are the symptoms of low vitamin E?

The body needs vitamin E to function, making it an essential vitamin. It is fat-soluble, meaning that it requires fat from the diet to be properly absorbed. Vitamin E is mainly stored in the liver before being released into the blood stream for use.

Deficiency is uncommon and typically the result of an underlying condition. Some premature infants also have low levels.
Vitamin E occurs in eight chemical forms. With a blood test, a doctor can learn how much of one form, alpha-tocopherol, a person has. Using this information, they can determine whether a person's overall level of vitamin E.
A normal level is usually with the range of 5.5–17 milligrams per liter (mg/L). The normal range may be different for premature infants and children under 17. Normal ranges can also vary slightly among labs.
When an adult has less than 4 mg/L of vitamin E in their blood, they usually require supplementation.

Signs and symptoms of deficiency

Senior man outdoors massaging temples because of headache, disorientation, and vision problems.
Vitamin E deficiency may cause disorientation and vision problems.
Low levels of vitamin E can lead to:
  • Muscle weakness: Vitamin E is essential to the central nervous system. It is among the body's main antioxidants, and a deficiency results in oxidative stress, which can lead to muscle weakness.
  • Coordination and walking difficulties: A deficiency can cause certain neurons, called the Purkinje neurons, to break down, harming their ability to transmit signals.
  • Numbness and tingling: Damage to nerve fibers can prevent the nerves from transmitting signals correctly, resulting in these sensations, which are also called peripheral neuropathy.
  • Vision deterioration: A vitamin E deficiency can weaken light receptors in the retina and other cells in the eye. This can lead to loss of vision over time.
  • Immune system problems: Some research suggests that a lack of vitamin E can inhibit the immune cells. Older adults may be particularly at risk.
Muscle weakness and difficulties with coordination are neurological symptoms that indicate damage to the central and peripheral nervous systems.
The peripheral system is the network of nerves located beyond the brain and spinal cord. These neurons pass messages throughout the body.
The central nervous system communicates between the brain and the spinal cord.
The sheaths of neurons are mostly composed of fats. When the body has too little vitamin E, it contains fewer antioxidants that protect these fats, and the function of the nervous system breaks down.

Causes of vitamin E deficiency

- Genetics

Vitamin E deficiency often runs in families.
Learning about family history can make diagnosing certain rare, inherited diseases easier. Two of these diseases, congenital abetalipoproteinemia and familial isolated vitamin E deficiency, are chronic and result in extremely low vitamin E levels.

- Medical conditions

Vitamin E deficiency can also result from diseases that severely reduce the absorption of fat. This is because the body requires fat to absorb vitamin E correctly.
Some of these diseases include:
Deficiency is also common in newborns and babies born prematurely who have lower birth weights and less fat.
Premature infants are at particular risk because an immature digestive tract can interfere with fat and vitamin E absorption.
Vitamin E deficiencies in these infants can also lead to hemolytic anemia, which destroys red blood cells.

When to see a doctor

When a person has no history of genetic disease but experiences any symptoms of a vitamin E deficiency, they should contact a doctor.
An extremely low level of vitamin E in the blood can indicate an underlying health issue. Further testing will help to determine the cause and treatment options.

What are the treatment options?

Doctor holding newborn baby.
Newborns and premature babies may be given a vitamin E supplement via a tube in the stomach.
Vitamin E supplementation is often effective.
Newborns and premature babies
Current practice involves providing vitamin E supplementation through a tube in the stomach. When necessary, it can also be administered intravenously.
While one dose can sufficiently raise blood levels of vitamin E, multiple doses may be required.
Children and adults
Children and adults with deficiencies caused by inherited conditions require supplementation with high doses of vitamin E.
Supplementation can stop the progression of the disease. When the deficiency is detected early, it may prevent neurological symptoms.

source : Medical News Today

Wednesday, February 28, 2018

Hidden Secret of Immortality Enzyme Telomerase .Can we stay young forever, or even recapture lost youth?

Research from the laboratory of Professor Julian Chen in the School of Molecular Sciences at Arizona State University recently uncovered a crucial step in the telomerase enzyme catalytic cycle. This catalytic cycle determines the ability of the human telomerase enzyme to synthesize DNA "repeats" (specific DNA segments of six nucleotides) onto chromosome ends, and so afford immortality in cells. Understanding the underlying mechanism of telomerase action offers new avenues toward effective anti-aging therapeutics. illustration depicting the enzyme telomerase This figure depicts the enzyme telomerase as well as telomeres relative to a chromosome.
Typical human cells are mortal and cannot forever renew themselves. As demonstrated by Leonard Hayflick a half-century ago, human cells have a limited replicative lifespan, with older cells reaching this limit sooner than younger cells. This "Hayflick limit" of cellular lifespan is directly related to the number of unique DNA repeats found at the ends of the genetic material-bearing chromosomes. These DNA repeats are part of the protective capping structures, termed "telomeres," which safeguard the ends of chromosomes from unwanted and unwarranted DNA rearrangements that destabilize the genome.
Each time the cell divides, the telomeric DNA shrinks and will eventually fail to secure the chromosome ends. This continuous reduction of telomere length functions as a "molecular clock" that counts down to the end of cell growth. The diminished ability for cells to grow is strongly associated with the aging process, with the reduced cell population directly contributing to weakness, illness, and organ failure.
The fountain of youth at molecular level
Counteracting the telomere shrinking process is the enzyme, telomerase, that uniquely holds the key to delaying or even reversing the cellular aging process. Telomerase offsets cellular aging by lengthening the telomeres, adding back lost DNA repeats to add time onto the molecular clock countdown, effectively extending the lifespan of the cell. Telomerase lengthens telomeres by repeatedly synthesizing very short DNA repeats of six nucleotides -- the building blocks of DNA -- with the sequence "GGTTAG" onto the chromosome ends from an RNA template located within the enzyme itself. However, the activity of the telomerase enzyme is insufficient to completely restore the lost telomeric DNA repeats, nor to stop cellular aging.
The gradual shrinking of telomeres negatively affects the replicative capacity of human adult stem cells, the cells that restore damaged tissues and/or replenish aging organs in our bodies. The activity of telomerase in adult stem cells merely slows down the countdown of the molecular clock and does not completely immortalize these cells. Therefore, adult stem cells become exhausted in aged individuals due to telomere length shortening that results in increased healing times and organ tissue degradation from inadequate cell populations.
Tapping the full potential of telomerase
Understanding the regulation and limitation of the telomerase enzyme holds the promise of reversing telomere shortening and cellular aging with the potential to extend human lifespan and improve the health and wellness of elderly individuals. Research from the laboratory of Chen and his colleagues, Yinnan Chen, Joshua Podlevsky and Dhenugen Logeswaran, recently uncovered a crucial step in the telomerase catalytic cycle that limits the ability of telomerase to synthesize telomeric DNA repeats onto chromosome ends.
"Telomerase has a built-in braking system to ensure precise synthesis of correct telomeric DNA repeats. This safe-guarding brake, however, also limits the overall activity of the telomerase enzyme," said Professor Chen. "Finding a way to properly release the brakes on the telomerase enzyme has the potential to restore the lost telomere length of adult stem cells and to even reverse cellular aging itself."
This intrinsic brake of telomerase refers to a pause signal, encoded within the RNA template of telomerase itself, for the enzyme to stop DNA synthesis at the end of the sequence 'GGTTAG'. When telomerase restarts DNA synthesis for the next DNA repeat, this pause signal is still active and limits DNA synthesis. Moreover, the revelation of the braking system finally solves the decades-old mystery of why a single, specific nucleotide stimulates telomerase activity. By specifically targeting the pause signal that prevents restarting DNA repeat synthesis, telomerase enzymatic function can be supercharged to better stave off telomere length reduction, with the potential to rejuvenate aging human adult stem cells.
Human diseases that include dyskeratosis congenita, aplastic anemia, and idiopathic pulmonary fibrosis have been genetically linked to mutations that negatively affect telomerase activity and/or accelerate the loss of telomere length. This accelerated telomere shortening closely resembles premature aging with increased organ deterioration and a shortened patient lifespan from critically insufficient cell populations. Increasing telomerase activity is the seemingly most promising means of treating these diseases.
While increased telomerase activity could bring youth to aging cells and cure premature aging-like diseases, too much of a good thing can be damaging for the individual. Just as youthful stem cells use telomerase to offset telomere length loss, cancer cells employ telomerase to maintain their aberrant and destructive growth. Augmenting and regulating telomerase function will have to be performed with precision, walking a narrow line between cell rejuvenation and a heightened risk for cancer development.
Distinct from human stem cells, somatic cells constitute the vast majority of the cells in the human body and lack telomerase activity. The telomerase deficiency of human somatic cells reduces the risk of cancer development, as telomerase fuels uncontrolled cancer cell growth. Therefore, drugs that increase telomerase activity indiscriminately in all cell types are not desired. Toward the goal of precisely augmenting telomerase activity selectively within adult stem cells, this discovery reveals the crucial step in telomerase catalytic cycle as an important new drug target. Small molecule drugs can be screened or designed to increase telomerase activity exclusively within stem cells for disease treatment as well as anti-aging therapies without increasing the risk of cancer.

Source : Arizona State University

Largest study of its kind finds alcohol use biggest risk factor for dementia

Alcohol use disorders are the most important preventable risk factors for the onset of all types of dementia, especially early-onset dementia. This according to a nationwide observational study, published in The Lancet Public Health journal, of over one million adults diagnosed with dementia in France.
This study looked specifically at the effect of alcohol use disorders, and included people who had been diagnosed with mental and behavioural disorders or chronic diseases that were attributable to chronic harmful use of alcohol.
Of the 57,000 cases of early-onset dementia (before the age of 65), the majority (57%) were related to chronic heavy drinking.
The World Health Organization (WHO) defines chronic heavy drinking as consuming more than 60 grams pure alcohol on average per day for men (4-5 Canadian standard drinks) and 40 grams (about 3 standard drinks) per day for women.
As a result of the strong association found in this study, the authors suggest that screening, brief interventions for heavy drinking, and treatment for alcohol use disorders should be implemented to reduce the alcohol-attributable burden of dementia.
"The findings indicate that heavy drinking and alcohol use disorders are the most important risk factors for dementia, and especially important for those types of dementia which start before age 65, and which lead to premature deaths," says study co-author and Director of the CAMH Institute for Mental Health Policy Research Dr. J├╝rgen Rehm. "Alcohol-induced brain damage and dementia are preventable, and known-effective preventive and policy measures can make a dent into premature dementia deaths."
Dr. Rehm points out that on average, alcohol use disorders shorten life expectancy by more than 20 years, and dementia is one of the leading causes of death for these people.
For early-onset dementia, there was a significant gender split. While the overall majority of dementia patients were women, almost two-thirds of all early-onset dementia patients (64.9%) were men.
Alcohol use disorders were also associated with all other independent risk factors for dementia onset, such as tobacco smoking, high blood pressure, diabetes, lower education, depression, and hearing loss, among modifiable risk factors. It suggests that alcohol use disorders may contribute in many ways to the risk of dementia.
"As a geriatric psychiatrist, I frequently see the effects of alcohol use disorder on dementia, when unfortunately alcohol treatment interventions may be too late to improve cognition," says CAMH Vice-President of Research Dr. Bruce Pollock. "Screening for and reduction of problem drinking, and treatment for alcohol use disorders need to start much earlier in primary care." The authors also noted that only the most severe cases of alcohol use disorder -- ones involving hospitalization -- were included in the study. This could mean that, because of ongoing stigma regarding the reporting of alcohol-use disorders, the association between chronic heavy drinking and dementia may be even stronger.

Source :  Centre for Addiction and Mental Health


Every day, your skin goes to battle with the environment. UV rays, pollution, blue light from laptops, and harsh ingredients do a number on our skin—which is why the next wave of skin-care products is working to support it in the modern world. Think “skin-barrier-supporting” and “microbiome-enhancing” serums that help fuel optimal skin function and, of course, a healthy glow.
“Especially now, there are lots of impacts from pollutants in the environment and ozone to UV rays, and all of these can have a negative impact on the [skin’s] lipid barrier,” explains Jim Hammer, a cosmetic chemist, such as redness, acne, or dryness. “So, proper skin barrier function is really key to maintaining healthy, hydrated skin.”
Beauty brands are rising to the occasion with new products designed to nourish and support this important ecosystem of the skin, says Cindy DiPrima Morisse, co-founder of CAP Beauty. “Instead of stripping away layers of the skin, products are helping build up its own defenses.”
Launches from companies like Marie Veronique, Biossance, Allies of Skin, and microbiome-protecting Mother Dirt are anticipating the environmental stressors your skin might encounter over the day, and the imbalances they could cause.
They’re using ingredients like lipids, ceramides, adaptogens, and even live bacteria to help fortify your skin’s natural functioning, so it can do its thing—and be your first natural line of defense against technology and the pollutants of the modern world.

Source :


Buzz about turmeric’s anti-inflammatory powers dominated 2017, but 2018 is all about moringa. North American healthy food companies have discovered this super-green from south of the equator, and it’s being touted as an even more powerful inflammation fighter.
Called the “world’s most useful tree” by scientists, moringa’s healing history in India, Chile, and Africa has been little known in this part of the world. But its nutrition profile is now becoming super brag-worthy on the wellness scene: It has twice the protein of spinach and three times as much iron, says nutritionist (and Well+Good Council member) Kimberly Snyder. According to the journal Ecology of Food and Nutrition, it has impressive amounts of calcium, potassium, and vitamin A. So this is pretty much a Survivor Island superfood of choice.
Fortunately, the buzzy veggie has a likable taste—its seeds are slightly sweet, and its ground leaves are nutty and grassy like tea. So food companies are not wasting a minute adding moringa into everything from nutrition bars and protein powders (Kuli Kuli) to juices (Pressed Juicery) and kombucha (as in,Suja’s new adaptogenic brew). Moringa’s even entering the chip aisle, where you can grab a bag of Vegan Rob’s Moringa Puffs for your next Netflix girls’ night in.
Oh, and in 2018 expect beauty brands featuring super-nourishing moringa oil (which can be a fair-trade ingredient) to make a point of telling you how great it is for your skin. Based on our research, they might be right.
source :

Friday, January 20, 2017

The Fluoride Poisoning Antidote

Image result for poisoning sickRegulatory agencies still haven’t budged on their official position that water fluoridation is a wonderfully beneficial public health policy with no inherent risks.

 Meanwhile, a tidal wave of anti-fluoride research and activism continues to build—there’s now such an abundance of evidence illustrating fluoride’s toxicity that the position of policy makers seems either laughable or outright deceitful. The EPA—which now lists fluoride as a neurotoxin—is the one exception (though their opinion doesn’t seem to be having much effect on actual policy).
Over 100 studies have conclusively linked fluoride to cognitive dysfunction, endocrine disruption, childhood development issues, and many other health problems.[1] For this reason, nearly a dozen countries around the world have already banned water fluoridation, and many U.S. cities and counties have also rejected it. 
If you live in a place with integrated access to unfluoridated water, you’re among the lucky few. If the rest of us wish to protect ourselves from the ravages of fluoride toxicity, we need to take measures into our own hands.
So what’s the solution?
The obvious move is to remove fluoride from your life by whatever measures possible. This can be rather tricky, though—water filters will not remove fluoride unless they’re specifically designed to do so (none of the most popular mainstream products come equipped with this feature, so be sure to seek it out specifically). Fluoride toothpaste is also a no-no, and even showering without a filter can be hazardous without purchasing yet another specialty filter.
This study is ground-breaking for two reasons: it provides further rock-solid evidence that fluoride is highly toxic, and it shows that turmeric is an incredibly promising “antidote” for this widespread poison.
Research protocol requires that study hypotheses are presented with great rigor and without leaps in logic—so it’s a huge deal that this study posits right at the start that “fluoride toxicity through drinking water is well-recognized as a global problem,” and that “health effect reports on fluoride exposure include various cancers, adverse reproductive activities, cardiovascular, and neurological diseases.”[2]  
Turmeric is well-known for its remarkable brain-supporting abilities. It’s a powerful antioxidant and anti-inflammatory agent, and has even been used with great success to treat Alzheimer’s patients. This is one of the first attempts to quantity its specific effects on fluoride-induced brain damage (that’s right: your drinking water may be giving you brain damage).
For this study, researchers measured neurotoxicity with a fittingly named marker known as brain rancidity. This state describes a type of oxidative stress in the brain called lipid peroxidation, which dramatically accelerates the creation of highly damaging free radicals.
They measured changing levels of brain rancidity across four groups of mice: one given no fluoride or curcumin (one of active components in turmeric), one given only fluoridated water, one given fluoridated water and curcumin, and one given only curcumin.
The results were remarkable: mice given only fluoridated water exhibited dramatically increased levels of brain rancidity, but the mice also given curcumin maintained very near normal, non-toxic levels. Curcumin was able to neutralize all the potential damage!
Researchers attribute this effect to turmeric’s unrivaled capacity to protect against a wide variety of oxidative free radicals, to control inflammation (in the brain and throughout the body), and to boost the body’s production of its own antioxidants.
Just one of many reasons to become friends with turmeric
Because it targets so many of the fundamental factors behind disease and toxicity, turmeric has been proven helpful for nearly every health problem imaginable. And because fluoride toxicity is such a looming risk for the vast majority of Americans, turmeric’s ability to counteract it is just one more reason why everyone can benefit from integrating turmeric into his or her life.
It’s important to use turmeric in the right way, though, if you want to receive its therapeutic benefits to the full extent. In the study cited above, curcumin was dissolved in olive oil, because binding turmeric alkaloids to a fatty substance creates an emulsion that is more readily absorbed by the body.
A process called liposomal delivery takes this one step further by completely merging turmeric compounds with a phospholipid to create a liposome. Liposomes ensure that turmeric is absorbed up to twenty times more completely than regular turmeric. So if you’re really serious about using turmeric to protect yourself against fluoride toxicity, make sure to pick up a liposomal turmeric extract, like this one from Purathrive.
The slowness with which policy makers are responding to the dire need to ban water fluoridation is incredibly frustrating—but by taking a liposomal turmeric extract on a daily basis, you can ensure that the health of your brain (and your entire body) is safeguarded.

Thursday, January 5, 2017

Eating Habits That Contribute to a Fatty Liver

According to studies, your diet is closely tied in with the creation of fatty liver. That's what researchers worldwide are reporting in their research. Thus, by reversing your eating habits that are out of alignment with healthy eating, you can then prevent and reverse fatty liver disease. Fatty liver disease is reversible if you attack it with good nutrition in its early stages.
Below is a list of 11 eating habits to make sure you are already including in your daily routines:
1 Stop eating processed fats and hydrogenated fats.
Check the labels of the foods you are eating; when you see hydrogenated fats on the list, discard the food. It's contributing to the promotion of a fatty liver.
2 Replace your vegetable oils with olive oil and coconut oil.
The most easily oxidized oils are the vegetable oils (corn, safflower, vegetable, soy, canola) because they are not stable; their chemical composition makes them subject to breakdown by light and heat. Eliminating these oils from your diet helps preserve your antioxidant levels.
3 Stop eating sugar and sugary foods.
Sugar affects your immunity, your metabolism, and your blood sugar levels. It also is a sure way to gain weight. When blood sugar levels aren't stable, metabolic syndrome occurs. Metabolic syndrome is tied closely with fatty liver disease.
4 If you're drinking coffee, make sure it's organic.
Non-organic coffee is loaded with chemicals and pesticides. Non-organic decaffeinated coffee is additionally loaded with nickel. The more toxins you consume, the harder you make your liver work.
5 Eat healthy protein foods, in serving sizes.
Eating 4 ounces protein (chicken, fish, beef, buffalo, wild meats, pork, poultry) twice daily is not excessive. Don't fear red meat; fear only the overconsumption of commercially grown red meats. When cattle consume a diet based on corn, it alters the composition of the fats in the meat. However, there is nothing wrong with grass fed beef.
6 Take supplements that correct any nutritional deficiencies you might have.
Nutritional deficiencies, such as methionine, choline, vitamin A, vitamin C, vitamin D, and vitamin E are tied to fatty liver. You need all vitamins and minerals, not just some of them. Take a good program of supplements.
7 Make sure antioxidants are amongst your current supplements.
The antioxidants vitamin C, E and A have been shown in the research to be medically important to prevent and reverse fatty liver.
8 Stop the alcohol.
Alcoholic drinks cause fatty liver disease.
9 Stop eating rancid nuts, especially peanuts.
Nuts that are rancid often contain the mold aspergillus, which creates the toxin aflatoxin. Peanuts are notorious for their high content of aflatoxin. Aflatoxin kills liver cells.
10 Don't overeat foods with saturated fats.
Foods with saturated fats are more stable in the body than unsaturated (vegetable oils) but you can still eat too many of them. Don't overeat them.
11 Avoid the salty foods.
High amounts of salt place an extra burden on the body and especially the liver. You need a pinch of salt each day, but not much more than that. 

Sunday, January 1, 2017

This Could Improve Chemotheraphy

This year alone, it’s estimated that nearly 600,000 people will die from cancer[1]—to say that the disease is still a prevalent and persistent issue is an enormous understatement.
 Despite huge expenditures of time and money, our society and medical system still hasn’t quite figured out how to stand up to cancer. While a number of alternative therapies show great promise, they’re still crowded out and derided in favor of conventionally accepted treatment modalities.
 Chemotherapy is one of the most widely practiced oncological treatments, despite its numerous drawbacks. Research reveals that, for some cancers, chemotherapy’s contribution to overall survival and recovery rates is shockingly small,[2] especially given the horrendous side effects that it can cause. These success rates, even in the case of cancers against which chemotherapy is more effective, barely outweigh its risks, according to some experts.
 All that being said, however, it’s still an option chosen by many people battling cancer. While it’s becoming increasingly clear that there are much better and safer ways to treat cancer, the draw of mainstream medicine is strong.

 Many patients are ostracized by their family and friends for seeking alternative treatments, and conventional oncologists can be notoriously skeptical and unsupportive of trying any protocols not “sanctioned” by the cancer industry. In some states, it’s even illegal for doctors to give (or even talk about) alternative cancer treatments.[3]
 If you or a loved one has already made the decision to undergo chemotherapy, though, the best thing you can do is to optimally support the process. Stay positive, and offer emotional support for whatever decision has been made. Most importantly, take measures that can minimize the risks of chemotherapy while maximixing its efficacy.
 A wealth of studies demonstrates that turmeric is a safe and effective natural remedy for accomplishing this task. Let’s take a look at some research-backed reasons why it’s wise for everyone undergoing chemotherapy to integrate turmeric into their treatment protocol.
 Patients obviously only willfully undergo the hardships of chemotherapy because they want it to get rid of their cancer—there’s nothing worse than having to endure the exorbitant cost and frightening side effects of chemo, all for a less than inspiring chance at full recovery.
 Chemotherapy already doesn’t have an overly impressive success rate, and it’s made even worse by multidrug impediment, wherein cancer cells become resistant to one or more of the drugs comprising a patient’s chemotherapy cocktail.
 To minimize this issue, which researchers have called “a major impediment to effective chemotherapy,” the medical community has begun the search for chemosensitizers—that is, low-impact therapeutic adjuncts that make patients’ bodies more sensitive to chemotherapy drugs (and thus allow the treatment to be more effective).[4]
 A landmark study published in the journal Anticancer Research demonstrated that curcumin (one of the beneficial compounds in turmeric) effectively sensitizes drug-resistant cancer cells to chemotherapy drugs.[5] Other studies confirmed that this mechanism works against a variety of cancer cells, including breast cancer,[6] esophageal cancer,[7] colon cancer,[8] stomach cancer,[9] and pancreatic cancer.[10]
 It carries out this feat by altering the genetic coding of proteins that control resistance in cancer cells, by damaging the DNA of cancer cells (while leaving healthy cells untouched), and by disrupting the signaling pathways necessary for cancer cell maintenance and proliferation.
 And that’s just the beginning—turmeric uses plenty of other tricks that directly combat cancer, with the end result that the chemotherapy patient has a better chance of recovery. Curcumin helps the body’s own defenses fight the spread of the disease,[11] induces apoptosis (programmed cell death) in cancer cells,[12] and can even slow or halt the regeneration of cancer stem cells (the drug-resistant “mother cells” that control that growth of tumors).[13]
 It’s because of these incredible healing mechanisms that turmeric could redefine the way we treat cancer. While chemotherapy and other risky treatment methods remain standard, though, turmeric can at least help can the protocol as safe and effective as possible.
 But here’s the catch: as we’ve discussed in our other articles, it’s very difficult for the body to absorb curcumin and the other beneficial alkaloids in turmeric. Even a 95% standardized extract of curcumin will only be 1% bioavailable—so obviously, raw turmeric root or powder is negligibly beneficial when used therapeutically for cancer or other severe illness.
 Researchers testing the anti-cancer properties of turmeric always use a formulation method that increases the bioavailability of curcumin (usually some kind of nano-encapsulation method that binds turmeric alkaloids to lipids, thus allowing the compounds to pass directly through cell walls).
 Liposomal turmeric is one of the leading types of formulations—curcumin is a lipophilic (fat-loving) molecule, so it lends itself perfectly to this delivery method.

Friday, December 30, 2016

What Goes In A Fatty Liver Disease Diet

As a Fatty Liver Disease patient, you may now be aware of the necessary changes that should be done in terms of your lifestyle and activity. The most basic thing is to quit your vices—most especially, smoking and drinking.
Keeping up with a healthy and active lifestyle can be a challenge, especially for the uninitiated. However, don’t give in to the pressure. You can achieve and get used to the healthy way if you know how to take it slowly, but surely.
Of course, this article is made to help you out with this new, but healthy phase in your life.
Let’s start with the basics, shall we?
This table contains the recommended daily intake for Fatty Liver/Non-fatty Liver disease patients:
Daily Intake1,200-1,500 kcal
Daily Fat IntakeLess than 35% of daily intake
Daily Carbohydrate & Protein IntakeLesser or at least 50% of daily intake (More on the types of food later).
Recommended Physical ActivityAt least 150 minutes/week for moderate to vigorous exercises.
It looks so simplified when it’s viewed in that table, eh? Now comes the heavy part—how to implement it.
As you can see, the recommended daily intake to start your Fatty Liver Disease diet is just around 1,200-1,500 kcal every day. That’s actually close to the usual recommended daily intake, so you won’t be actually starving yourself when it comes to this type of diet. The main thing to look out for is choosing the right kind of food and beverage to not stray from your number of required kcal each day.
It’s also important to note that you need fats, protein and carbohydrate in your diet. So again, you are very much welcome to eat meat and possibly, a few treats. All you need now is control.
First and foremost, let’s strike out the types of food that are too heavy in terms of kcal, and can be unhealthy for your overall health. Below is a list:
  • Processed Foods (that includes any frozen or pre-processed food)
Well, this one is pretty much hard to avoid since the market is teeming with packed and processed stuff. The main thing to remember though is to opt for fresh and organic fruits—don’t buy in the frozen section anymore, or any food that’s canned or sealed with preservatives.
  • Sweet, Sugary Treats
This is particularly bad for Fatty Liver Disease patients who are also diabetic at the same time. But it’s still pretty bad for Fatty Liver Disease patients without diabetes because high sugar foods can overwork the liver.
  • Sodas
Since Sodas contain high doses of sugar, then it’s a no.
  • Fastfood Basics and Pastry
Burger, fries, pizza, donuts and cakes. They may be a gastric treat, but the cholesterol and high calorie each of these foods possesses is not friendly to your liver—or to your overall health. More so, these foods are very much processed and laced with preservatives, so it’s a big DON’T.
Now the don’ts are already taken care of, here are the foods that you should eat:
  • Whole grains
These foods are high in fiber and can greatly help your liver get rid of all that bad fat and other toxins.
  • Lean Meat and Vegetable-based protein
Since you need protein in your body, it’s important to note that you still need to avoid the bad types of meat (like pork). For healthy protein sources, go for beans, fish and other types of seafood.
  • Fruits and vegetables
Of course this is the most important part of your diet, fruits and vegetables are very low in calories and rich in fiber and nutrients. So they are perfect for daily intake without you feeling guilty—no matter how much you chomp down on them. However, for people who are also diabetic or at risk of being one, it’s important to note that most fruits contain high amounts of sugar, so it won’t be advisable to eat too much. Other fruits such as lemon and lime however, don’t contain much sugar, so you can have those instead.
Here’s an example of breakfast, lunch, and dinner meals on a Fatty Liver Disease diet:
  • Whole grain oats
  • Wheat Bread
  • Natural Strawberry Jam
  • Natural Orange Juice
  • Roasted Chicken
  • Side dish of Steamed Veggies (Carrots and Broccoli)
  • Roasted Chicken
  • Side dish of Steamed Veggies (Carrots and Broccoli)
  • Salmon with Slices of Lemon
  • Green Beans

For more liver-friendly recipes and a month-long guide of food choices, you can
What If I Want to Eat Out?
It sometimes cannot be avoided that we have certain occasions to celebrate, and we do that by treating ourselves, our family, or colleagues to a lunch or dinner out of the house. But, is it okay? First, just avoid the fastfood chains since you may just be tempted to eat the unhealthy stuff.
Just go to a nice restaurant and if possible, just order a veggie salad or a seafood/fish dish for yourself. Pasta is good too!
Which Kind of Exercise Is Best?
If you are still starting out with a regular exercise program, don’t rush your body into a strenuous one. The purpose of your exercise is to burn excess fat, so just go with an activity that can make you at least sweat much. For starters, you can go for 10-20 minute walks or jogs. You shouldn’t also be on a rush to lose weight.
Since this is a “Diet”, Am I Required to Lose Weight?
Consequently, you are really bound to lose weight in a Fatty Liver Disease diet. However, it’s not really the main goal per se, because the goal here is to employ an active and healthy lifestyle that you can maintain easily for the rest of your life. The requirement here however is to monitor and keep watch of your weight gain or loss while you are on the diet. Don’t be on a rush to lose weight fast because it could worsen your Fatty Liver. Just make sure that you lose weight on a “healthy” scale, and that’s just at least 5-10% less of your overall weight per year when you start the diet.
What Other Alternatives I Can Take?
Refreshments! Specifically, you can make your own juices that can help the liver get rid all that excess fat and toxins in your body. That’s by detoxing! To cleanse your liver, you need to create and mix juices made from fruits or vegetables and drink it daily as part of your Fatty Liver Disease diet.
How can this help?
As mentioned earlier, fruits and veggies are high in fiber, so they are your helpers when it comes to cleaning your liver. Juicing just gives it an added benefit—it’s yummy and refreshing (and of course, healthy)! For starters, you can try out a juice made from lemon, cucumber and mint leaves. For more juicing recipes, you can go here.